Body obtained after coagulation of blood, half-solid consistency, spongy nature (like a sponge) and consists of a variety of protein: the fibrin in the blood. The clot's role is to retain in its meshes, platelets and red blood cells (RBCs) at the origin of its color very dark red.
The term thrombosis means in turn the formation of a clot (thrombus) in a blood vessel or one of the chambers of the heart in living systems. For some authors this term also refers todisorder that leads to thrombosis.
By extension, the term of thrombosis of choledochal bile Chauffard applies to the formation of large stone that occur within the common bile duct (channel flow in which the bile to the duodenum and confer). The process leading to the formation of this large calculation is as follows: deposition of successive layers of a mixture of cholesterol and bile pigments.
When fresh blood is collected and then brought into contact with air, it quickly turns into a kind of mass, after a few hours, eventually retract and let out (by sweating) a liquid serum which is the liquid part of blood. Indeed, the formed elements (white blood cells, red blood cells and platelets) were retained within the clot and the liquid serum (serum) is the clot on its surface.
The term thrombosis means in turn the formation of a clot (thrombus) in a blood vessel or one of the chambers of the heart in living systems. For some authors this term also refers todisorder that leads to thrombosis.
By extension, the term of thrombosis of choledochal bile Chauffard applies to the formation of large stone that occur within the common bile duct (channel flow in which the bile to the duodenum and confer). The process leading to the formation of this large calculation is as follows: deposition of successive layers of a mixture of cholesterol and bile pigments.
When fresh blood is collected and then brought into contact with air, it quickly turns into a kind of mass, after a few hours, eventually retract and let out (by sweating) a liquid serum which is the liquid part of blood. Indeed, the formed elements (white blood cells, red blood cells and platelets) were retained within the clot and the liquid serum (serum) is the clot on its surface.
The slowing circulation, often the result of a sedentary lifestyle exaggerated. The decay of the vessels and more specifically in the intima, which is to say, the inner wall of the vessels, due to infection or deposits of atherosclerosis (fatty substances). The increase in blood viscosity (decreased hematocrit). In the plasma coagulation Gilbert and Weil (abnormal blood clotting), there is a delay of coagulation, red blood cells have time to settle. The clot then presents a kind of defect, since its upper part, whitish transparent, contains platelets and white blood cells that retract more. It includes a second lower red color more opaque than normal clot. Plasma coagulation for certain severe anemia and is also observed in hemophilia. Syndrome of disseminated intravascular coagulation Hardaway and McKay (in English: Disseminated intravascular coagulation syndrome), also known as DIC, defibrination syndrome, coagulopathy consumption Lasch, is a collection of symptoms characterized by disturbances of blood coagulation due to the disappearance of fibrinogen from the circulating blood. The syndrome of disseminated intravascular coagulation is the result of the sudden onset of activation factors of thrombin leading to fibrin deposition in microvessels leading to their obliteration by thrombosis. They persist longer or shorter. Following this mechanism, the blood becomes incoagulable because it has consumed fibrinogen and other clotting factors (factor V and factor VIII) and platelets. This is the source of bleeding. The hypercoagulability (also called thrombophilia) corresponds to an increase in capacity of the blood to clot. Patients with hypercoagulability the following symptoms: increased speed of blood clotting (known hypercoagulable clotting), the test of tolerance to heparin in vitro and thromboelastography are disrupted, there is also increasing the resistance of the clot (in this case it is a structural hypercoagulability). Blood levels of fibrin and platelets in the blood is higher than normal (see below the lab). Other etiologies (origins) are grouped under the term platelet disorder (qv).
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